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• W2560805164 startingPage "9" @default.
• W2560805164 abstract "Advancing age is associated with progressive declines in physiological function that lead to overt chronic disease, frailty, and eventual mortality. Importantly, age-related physiological changes occur in cellularity, insulin-responsiveness, secretory profiles, and inflammatory status of adipose tissue, leading to adipose tissue dysfunction. Although the mechanisms underlying adipose tissue dysfunction are multifactorial, the consequences result in secretion of proinflammatory cytokines and chemokines, immune cell infiltration, an accumulation of senescent cells, and an increase in senescence-associated secretory phenotype (SASP). These processes synergistically promote chronic sterile inflammation, insulin resistance, and lipid redistribution away from subcutaneous adipose tissue. Without intervention, these effects contribute to age-related systemic metabolic dysfunction, physical limitations, and frailty. Thus adipose tissue dysfunction may be a fundamental contributor to the elevated risk of chronic disease, disability, and adverse health outcomes with advancing age." @default.
• W2560805164 created "2016-12-16" @default.
• W2560805164 creator A5008269407 @default.
• W2560805164 creator A5009102749 @default.
• W2560805164 creator A5019907813 @default.
• W2560805164 creator A5057209268 @default.
• W2560805164 date "2017-01-01" @default.
• W2560805164 modified "2023-10-11" @default.
• W2560805164 title "Physiological Aging: Links Among Adipose Tissue Dysfunction, Diabetes, and Frailty" @default.
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