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- W2563680362 abstract "HBx acts as a multifunctional regulator that modulates various cellular responses, which can lead to development and progression of hepatocellular carcinoma (HCC). Here, we show that the HBx protein is also local- ized to peroxisomes, and this increases cellular reactive oxygen species (ROS) to levels that are higher than when HBx is localized to other organelles. The elevated ROS strongly activated nuclear factor (NF)-jB. In addition, the peroxisome-localized HBx increased the expressions of matrix metalloproteinases and decreased the expression of E-cadherin, which increased the invasive ability of HCC cells. Thus, a specific distribution of HBx to peroxisomes may contribute to HCC progression by increasing the invasive ability of HCC cells through elevation of the cellular ROS level." @default.
- W2563680362 created "2017-01-06" @default.
- W2563680362 creator A5079936146 @default.
- W2563680362 date "2014-01-01" @default.
- W2563680362 modified "2023-09-23" @default.
- W2563680362 title "Peroxisome-localized hepatitis Bx protein increases the invasion property of hepatocellular carcinoma cells" @default.
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