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- W2564116166 endingPage "2097" @default.
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- W2564116166 abstract "Apoptosis functions as a common mechanism to eliminate unnecessary or damaged cells during cell renewal and tissue development in multicellular organisms. More than 200 proteins constitute complex networks involved in apoptotic regulation. Imbalanced expressions of apoptosis-related factors frequently lead to malignant diseases. The biological functions of several apoptotic factors are manipulated through alternative splicing mechanisms which expand gene diversity by generating discrete variants from one messenger RNA precursor. It is widely observed that alternatively-spliced variants encoded from apoptosis-related genes exhibit differential effects on apoptotic regulation. Alternative splicing events are meticulously regulated by the interplay between trans-splicing factors and cis-responsive elements surrounding the regulated exons. The major focus of this review is to highlight recent studies that illustrate the influences of alternative splicing networks on apoptotic regulation which participates in diverse cellular processes and diseases." @default.
- W2564116166 created "2017-01-06" @default.
- W2564116166 creator A5000238307 @default.
- W2564116166 creator A5021402906 @default.
- W2564116166 creator A5037903596 @default.
- W2564116166 date "2016-12-14" @default.
- W2564116166 modified "2023-09-26" @default.
- W2564116166 title "Differential Impacts of Alternative Splicing Networks on Apoptosis" @default.
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