FRINK Linked Data Fragments server

Matches in SemOpenAlex for { <https://semopenalex.org/work/W2565087262> ?p ?o ?g. }

• W2565087262 endingPage "208" @default.
• W2565087262 startingPage "191" @default.
• W2565087262 abstract "Ultrasound (US) allows the non-invasive evaluation of morphological changes of kidney structure (by means of B-Mode) and patterns of renal and extrarenal vascularization (by means of color-Doppler and contrast-enhanced US). In hypertensive subjects it offers a relevant contribution to the diagnosis of early renal damage, acute or chronic nephropathies and nephrovascular disease. However, morphological changes are often detected late and non-specific and in recent years evidence has increased regarding the clinical relevance of renal resistive index (RRI) for the study of vascular and renal parenchymal renal abnormalities. RRI is measured by Doppler sonography in an intrarenal artery, as the difference between the peak systolic and end-diastolic blood velocities divided by the peak systolic velocity. At first RRI was proved to be a marker of renal disease onset and progression; later the influence of systemic vascular properties on RRI was shown and authors claimed its use as an independent predictor of cardiovascular risk rather than of renal damage. Indeed, renal vascular resistance is only one of several renal (vascular compliance, interstitial and venous pressure), and extrarenal (heart rate, pulse pressure) determinants that concur to determine RRI individual values but not the most important one. The clinical relevance of RRI measurement as a surrogate endpoint of specific renal damage or/and as surrogate endpoint of atherosclerotic diffuse vascular damage is still debated.To summarize, from the literature: (a) In hypertensives with normal renal function and no albuminuria, especially in younger people, RRI is an early marker of renal damage that is especially useful when hypertension and diabetes concur in the same subjects. In these subjects RRI could improve current clinical scores used to stratify early renal damage. In older subjects RRI increases in accordance with the increase in systemic vascular stiffness and, because of this close relationship, RRI is also a marker of systemic atherosclerotic burden and the role of renal determinants can weaken. The clinical relevance was not specifically investigated. (b) In transplant kidney and in chronic renal disease high (>0.80) RRI values can independently predict renal failure. The recent claim that systemic (pulse pressure) rather than renal hemodynamic determinants sustain this predictive role of RRI, does not significantly reduce this predictive role of RRI. (c) Doppler ultrasound allows diagnosis and grading of renal stenosis in both fibromuscolar dysplastic and atherosclerotic diseases. Moreover, by RRI assay Doppler ultrasound can indirectly measure the hemodynamic impact of renal artery stenosis on the homolateral kidney, by virtue of the stenosis-related decrease in pulse pressure. However, in elderly subjects with atherosclerotic renal artery stenosis coexisting renal diseases can independently increase RRI by the augmentation in renal vascular stiffness and tubulo-interstitial pressure and hidden changes due to renal artery stenosis." @default.
• W2565087262 created "2017-01-06" @default.
• W2565087262 creator A5086012384 @default.
• W2565087262 date "2016-01-01" @default.
• W2565087262 modified "2023-10-01" @default.
• W2565087262 title "Renal Ultrasound (and Doppler Sonography) in Hypertension: An Update" @default.
• W2565087262 cites W1606143691 @default.
• W2565087262 cites W1964159158 @default.
• W2565087262 cites W1970537005 @default.
• W2565087262 cites W1973620057 @default.
• W2565087262 cites W1982483134 @default.
• W2565087262 cites W1982533775 @default.
• W2565087262 cites W1982747261 @default.
• W2565087262 cites W1985446258 @default.
• W2565087262 cites W1987456946 @default.
• W2565087262 cites W1988333796 @default.
• W2565087262 cites W1990105837 @default.
• W2565087262 cites W1996441257 @default.
• W2565087262 cites W1999503243 @default.
• W2565087262 cites W1999610398 @default.
• W2565087262 cites W2015471621 @default.
• W2565087262 cites W2015815870 @default.
• W2565087262 cites W2029346078 @default.
• W2565087262 cites W2030910315 @default.
• W2565087262 cites W2031587261 @default.
• W2565087262 cites W2033030077 @default.
• W2565087262 cites W2043456331 @default.
• W2565087262 cites W2051288209 @default.
• W2565087262 cites W2053607854 @default.
• W2565087262 cites W2059751705 @default.
• W2565087262 cites W2065320445 @default.
• W2565087262 cites W2065486513 @default.
• W2565087262 cites W2067473753 @default.
• W2565087262 cites W2081887242 @default.
• W2565087262 cites W2081972249 @default.
• W2565087262 cites W2086238162 @default.
• W2565087262 cites W2086380315 @default.
• W2565087262 cites W2090045150 @default.
• W2565087262 cites W2090151521 @default.
• W2565087262 cites W2098768854 @default.
• W2565087262 cites W2099678862 @default.
• W2565087262 cites W2103562591 @default.
• W2565087262 cites W2111053980 @default.
• W2565087262 cites W2120486347 @default.
• W2565087262 cites W2122826224 @default.
• W2565087262 cites W2123037922 @default.
• W2565087262 cites W2126125759 @default.
• W2565087262 cites W2133416234 @default.
• W2565087262 cites W2133875723 @default.
• W2565087262 cites W2138074241 @default.
• W2565087262 cites W2141902714 @default.
• W2565087262 cites W2143481804 @default.
• W2565087262 cites W2147514283 @default.
• W2565087262 cites W2150791156 @default.
• W2565087262 cites W2155053121 @default.
• W2565087262 cites W2156264974 @default.
• W2565087262 cites W2157156014 @default.
• W2565087262 cites W2164239451 @default.
• W2565087262 cites W2168211849 @default.
• W2565087262 cites W2168587456 @default.
• W2565087262 cites W2170508439 @default.
• W2565087262 cites W2179304282 @default.
• W2565087262 cites W2228436754 @default.
• W2565087262 cites W2283516282 @default.
• W2565087262 cites W2312236630 @default.
• W2565087262 cites W2312362587 @default.
• W2565087262 cites W2319097872 @default.
• W2565087262 cites W2391712484 @default.
• W2565087262 cites W2414421788 @default.
• W2565087262 cites W2416188358 @default.
• W2565087262 cites W2437149908 @default.
• W2565087262 cites W2467350993 @default.
• W2565087262 cites W2488219382 @default.
• W2565087262 cites W4235823177 @default.
• W2565087262 cites W4248253870 @default.
• W2565087262 doi "https://doi.org/10.1007/5584_2016_170" @default.
• W2565087262 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/27966109" @default.
• W2565087262 hasPublicationYear "2016" @default.
• W2565087262 type Work @default.
• W2565087262 sameAs 2565087262 @default.
• W2565087262 citedByCount "40" @default.
• W2565087262 countsByYear W25650872622018 @default.
• W2565087262 countsByYear W25650872622019 @default.
• W2565087262 countsByYear W25650872622020 @default.
• W2565087262 countsByYear W25650872622021 @default.
• W2565087262 countsByYear W25650872622022 @default.
• W2565087262 countsByYear W25650872622023 @default.
• W2565087262 crossrefType "book-chapter" @default.
• W2565087262 hasAuthorship W2565087262A5086012384 @default.
• W2565087262 hasBestOaLocation W25650872622 @default.
• W2565087262 hasConcept C126322002 @default.
• W2565087262 hasConcept C141341695 @default.
• W2565087262 hasConcept C142225936 @default.
• W2565087262 hasConcept C159641895 @default.
• W2565087262 hasConcept C164705383 @default.
• W2565087262 hasConcept C2776174234 @default.
• W2565087262 hasConcept C2778319312 @default.
• W2565087262 hasConcept C2778653478 @default.

• next