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- W2565698575 endingPage "300" @default.
- W2565698575 startingPage "293" @default.
- W2565698575 abstract "Atrial fibrillation (AF) is the commonest arrhythmia in clinical practice and is associated with increased cardiovascular morbidity and mortality. Obstructive sleep apnea (OSA), a common breathing disorder, is an independent risk factor for AF. Several pathophysiological mechanisms, including apnea-induced hypoxia, intrathoracic pressure shifts, sympathovagal imbalance, atrial remodeling, oxidative stress, inflammation and neurohumoral activation have been implicated in the occurrence of AF in OSA patients. In addition, OSA has been shown to reduce success rates of antiarrhythmic drugs, electrical cardioversion and catheter ablation in AF. Effective prevention of obstructive respiratory events by continuous positive airway pressure ventilation (CPAP) reduces sympathovagal activation and recurrence of AF. The present review describes the relationship between OSA and AF, presents the pathophysiological mechanisms implicating OSA in AF occurrence, and provides an update of the potential therapeutic interventions for patients with OSA and AF." @default.
- W2565698575 created "2017-01-06" @default.
- W2565698575 creator A5026137541 @default.
- W2565698575 creator A5062900671 @default.
- W2565698575 date "2017-03-01" @default.
- W2565698575 modified "2023-10-01" @default.
- W2565698575 title "Obstructive sleep and atrial fibrillation: Pathophysiological mechanisms and therapeutic implications" @default.
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