Matches in SemOpenAlex for { <https://semopenalex.org/work/W2567486062> ?p ?o ?g. }
- W2567486062 abstract "Acute and chronic alcohol consumption increases the produc tion of reactive oxygen species (ROS), and enhances lipid peroxidation of lipids, proteins, and DNA. The mechanism by which alcohol causes cell injury is still not clear but a major role for ROS andlipidperoxidation-endproductsisconsidered.Manypathways have been suggestedto play a role on how ethanol induces a state of “oxidative stress”, including redox-state changes, acetaldehyde production, damage to mitochondria, membrane injury, apopto sis, ethanol-induced hypoxia, effects on the immune system and altered cytokine production, increased endotoxin levels and activation of Kupffer cells, mobilization of iron, modulation of the an tioxidant defense, particularly mitochondrial glutathione (GSH), one electron oxidation of ethanol to 1-hydroxy-ethyl radical, and induction of CYP2E1. These pathways are not exclusive of one another and it is likely that several, indeed many, systems con tributetotheabilityofethanoltoinduceastateofoxidativestress." @default.
- W2567486062 created "2017-01-06" @default.
- W2567486062 creator A5082064300 @default.
- W2567486062 date "2008-01-01" @default.
- W2567486062 modified "2023-09-24" @default.
- W2567486062 title "Estrés oxidativo hepatocitario y hepatopatía alcohólica" @default.
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