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• W2571157765 abstract "Mitochondrial dysfunction plays a pivotal role in Alzheimer's disease (AD), even before signs of AD pathology are evident. Our previous research has shown that oxygen treatment can improve cognitive function in AD model mice. To address whether oxygen treatment is beneficial to mitochondrial biology, we analyzed differential expressions of hippocampal mitochondrial proteins in AD model mice given supplementary oxygen. Numerous respiratory chain, Kreb's cycle, and glycolysis proteins were upregulated significantly after oxygen treatment, suggesting that oxygen therapy can alleviate mitochondrial damage. Furthermore, the treatment was associated with decreased expressions of some AD biomarkers, suggesting oxygen treatment to be a potential therapy for AD." @default.
• W2571157765 created "2017-01-13" @default.
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• W2571157765 date "2017-02-03" @default.
• W2571157765 modified "2023-09-27" @default.
• W2571157765 title "Mitochondrial Repair Effects of Oxygen Treatment on Alzheimer’s Disease Model Mice Revealed by Quantitative Proteomics" @default.
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• W2571157765 doi "https://doi.org/10.3233/jad-161010" @default.
• W2571157765 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/28059791" @default.
• W2571157765 hasPublicationYear "2017" @default.
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