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• W2574014770 abstract "The clinical significance of small intestinal damage caused by nonsteroidal anti-inflammatory drugs (NSAIDs) remains under-appreciated. It occurs with greater frequency than the damage caused by these drugs in the upper gastrointestinal tract, but is much more difficult to diagnose and treat. Although the pathogenesis of NSAID enteropathy remains incompletely understood, it is clear that bacteria, bile, and the enterohepatic circulation of NSAIDs are all important factors. However, they are also interrelated with one another. Bacterial enzymes can affect the cytotoxicity of bile and are essential for enterohepatic circulation of NSAIDs. Gram-negative bacteria appear to be particularly important in the pathogenesis of NSAID enteropathy, possibly through release of endotoxin. Inhib- itors of gastric acid secretion significantly aggravate NSAID enteropathy, and this effect is due to significant changes in the intestinal microbiome. Treatment with antibiotics can, in some circumstances, reduce the severity of NSAID enteropathy, but published results are inconsis- tent. Specific antibiotic-induced changes in the microbiota have not been causally linked to prevention of intestinal damage. Treatment with probiotics, particularly Bifido- bacterium, Lactobacillus, and Faecalibacteriaum pra- usnitzii, has shown promising effects in animal models. Our studies suggest that these beneficial effects are due to colonization by the bacteria, rather than to products released by the bacteria." @default.
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• W2574014770 date "2015-01-01" @default.
• W2574014770 modified "2023-09-26" @default.
• W2574014770 title "NSAID enteropathy and bacteria: a complicated relationship Stephanie D. SyerRory W. BlacklerRebeca MartinGiada de Palma • Laura RossiElena VerduPremek BercikMichael G. Surette • Anne AucouturierPhilippe LangellaJohn L. Wallace" @default.
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• W2574014770 hasPublicationYear "2015" @default.
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