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- W2575014971 abstract "Intermittent claudication, according to the Fontaine classification, is a classical symptom of stage II peripheralarterial occlusive disease (PAOD) of the lower limbs. It results from the progression of atherosclerosisleading to the narrowing and complete occlusion of arteries. It manifests with pain in the muscles of the lowerlimbs which forces the patients to interrupt their current activity. Supervised treadmill training is believed tolead to the most favourable outcomes in the form of improved pain-free walking distance and maximumwalking distance. The improvement in pain related to intermittent claudication and in functional performanceare probably the combined effect of various mechanisms in response to the exercise training. Themost important mechanisms include: improved skeletal muscle metabolism, favourable haemorheologic changes,delayed progression of atherosclerosis, peripheral blood flow adaptation, improved economics of walking,and changed perception of pain. The role of these mechanisms and their contribution to the improved functionalperformance in this group of patients are not unequivocally established and continue to be disputable.In this paper we review and discuss the mechanisms responsible for improving walking distance observedduring walking exercises in patients with intermittent claudication. Acta Angiol 2010; 16, 2: 49-66" @default.
- W2575014971 created "2017-01-26" @default.
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- W2575014971 date "2010-01-01" @default.
- W2575014971 modified "2023-09-23" @default.
- W2575014971 title "Mechanisms responsible for reducing intermittent claudication as a result of treadmill training" @default.
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