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- W2580422358 endingPage "376" @default.
- W2580422358 startingPage "359" @default.
- W2580422358 abstract "The inner lining of normal blood vessels is composed of endothelial cells that form a surface resistant to the adhesion of circulating platelets. In areas where the endothelium is altered, however, or at sites of vascular damage where subendothelial or other extracellular matrices are exposed, firm platelet attachment rapidly occurs. This is a critical initial step in hemostasis and thrombosis, as well as in infl ammatory and immunopathogenic responses,1 requiring the concerted interaction of matrix proteins with platelet receptors ultimately leading to platelet activation and aggregation. The hemostatic response to vascular injury is contingent on the extent of damage, the specific matrix proteins exposed, and flow conditions. In this chapter, we focus first on the mechanisms responsible for the initial interaction of platelets with thrombogenic surfaces associated with vascular and tissue trauma. We then discuss the secondary events that, pursuant to the initial adhesion of platelets, lead to thrombus propagation and stabilization by mechanisms that mostly depend on the formation of interplatelet contacts." @default.
- W2580422358 created "2017-02-03" @default.
- W2580422358 creator A5048582799 @default.
- W2580422358 creator A5084109223 @default.
- W2580422358 date "2007-01-01" @default.
- W2580422358 modified "2023-10-12" @default.
- W2580422358 title "Platelet Thrombus Formation in Flowing Blood" @default.
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