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- W2583151421 abstract "species studied. Knowing whether the hypothalamicDepartment of Neuroscience NPY network is a component of the normal–natural ap-The University of Florida Brain Institute petite transduction pathway is fundamental for under-University of Florida College of Medicine standingthebrain’scontrolofappetiteand body weight.Gainesville, Florida 32610 Evidence in favor of this implication is the abrupt in-crease inNPY secretion before mealtimeinthe paraven-tricular nucleus, one of the sites normally engaged ingenerating appetite-evoking signals. This heightenedFew human diseases have spurred more public interestsecretion of NPYismaintained and appetiteissustainedrecently than obesity and an abnormal rate of bodyfor as longas food is withheld (Kalra et al.,1991). On theweight gain, the most prevalent nutritional disorders inother hand,if NPY secretion is haltedorits availabilityatdeveloped, industrialized nations. Obesity has reachedtarget sites is curtailed experimentally, appetite sub-epidemicstatussince,according totheInstitute ofMed-sides despitea severelossinbody energy.Furthermore,icine, 59% of Americans are clinically obese or at leastcontinuous NPYreceptor activationreproduces thenor-20% above their ideal body weight. This disease is amajor contributor to the high rate of morbidity due to mal pattern of intermittent feeding in satiated rats with-increased risk of cardiovascular diseases, including out developing tolerance, and accelerates the rate ofstroke and hypertension, and diabetes. Whereas extra- body weight gain culminating in obesity that is indistin-neouscauses,suchasovereatingand/orreducedphysi- guishable from that produced experimentally or seencal activity, undoubtedly underlie the increased inci- in genetic models (Kalra and Kalra, 1996). Given thesedence of obesity, the notion that genetic abnormalities results, researcherswere surprisedtofind that a geneticarea contributingfactorgainedanunprecedentedboost knockout of NPY had no obvious body weight pheno-in the public interest with the cloning of the first major type. However, subsequent experiments have shownobesity gene (" @default.
- W2583151421 created "2017-02-10" @default.
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- W2583151421 date "1997-01-01" @default.
- W2583151421 modified "2023-09-24" @default.
- W2583151421 title "Appetite and Body Weight Regulation: Minireview Is It All in the Brain?" @default.
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