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• W2585531699 abstract "Abstract Atrioventricular nodal reentrant tachycardia (AVNRT) is the most common form of paroxysmal supraventricular tachycardia. The exact electroanatomic circuit responsible for AVNRT remains elusive. Current evidence suggests that dual atrioventricular nodal (AVN) pathway physiology constitutes the substrate for AVN reentry. The different atrial inputs to the AVN, rather than functional longitudinal dissociation within the compact AVN, represent the fast and slow pathways involved in the reentrant circuit. AVNRT can manifest in different forms depending on the anatomic substrate forming the anterograde and retrograde pathways incorporated in the reentry circuit. Traditionally, AVNRT has been classified into “typical” or “atypical” forms. Typical AVNRT (anterograde slow-retrograde fast) accounts for 90% of AVNRTs. Atypical AVNRT variants are traditionally subclassified as either fast-slow or slow-slow types. Maintenance of AVNRT is dependent on AVN conduction; hence, maneuvers or drugs that slow AVN conduction and prolong AVN refractoriness are used to terminate the tachycardia. For chronic management of AVNRT, pharmacological therapy and catheter ablation can be considered. Long-term pharmacological therapy (including beta blockers and calcium channel blockers) can be effective in 30% to 60% of patients. On the other hand, catheter ablation is associated with higher efficacy (>95%) and low incidence of complications and therefore has become the preferred initial therapeutic approach. The slow pathway is the target of ablation for all variants of AVNRT. Initially, the rightward inferior AVN extension is targeted. Then, the leftward inferior AVN extension is targeted if needed." @default.
• W2585531699 created "2017-02-10" @default.
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• W2585531699 date "2009-01-01" @default.
• W2585531699 modified "2023-09-23" @default.
• W2585531699 title "Atrioventricular Nodal Reentrant Tachycardia" @default.
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• W2585531699 doi "https://doi.org/10.1016/b978-1-4160-5998-1.00016-1" @default.
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