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- W2589280134 abstract "Chromosomal translocations at 2p23 cause overexpression of anaplastic lymphoma kinase (ALK), a receptor tyrosine kinase involved in signalling pathways that regulate cell proliferation. This translocation occurs in 5% of lung adenocarcinoma and has been demonstrated to be useful as a therapeutic target for crizotinib. sinonasal adenocarcinomas (SNAC) are histologically similar to lung adenocarcinomas; the aim of this study was to evaluate the presence of ALK alterations in SNAC.Break-apart fluorescent in-situ hybridization was used to analyse the presence of ALK translocations in 96 tumour samples. In addition, ALK protein expression was studied by immunohistochemistry.The samples of SNAC did not show ALK translocation. Moreover, ALK protein expression was absent in all cases.These results suggest that ALK is not involved in SNAC." @default.
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- W2589280134 date "2017-01-01" @default.
- W2589280134 modified "2023-10-16" @default.
- W2589280134 title "Ausencia de translocaciones cromosómicas y de expresión proteica de ALK en los adenocarcinomas nasosinusales" @default.
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- W2589280134 doi "https://doi.org/10.1016/j.otorri.2016.02.003" @default.
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