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- W2592518628 endingPage "354" @default.
- W2592518628 startingPage "341" @default.
- W2592518628 abstract "Many cytokines and chemokines derived from variable liver cells are involved in the development of hepatic injury. Although these paracrine/autocrine mediators constitute exquisite networks to maintain the homeostatic balance in the liver, their derangements enhance liver failure and fibrosis. Together with experimental studies, analyses of circulating cytokines are important for determining their pathophysiological roles in human hepatic injuries. As hepatic macrophages are key players that mobilize various cytokines and chemokines in health and disease, we should also take their heterogeneity into account to understand the cytokine-related liver changes. In acute hepatic failure, excessive production of pro- and antiinflammatory cytokines results in toxic hypercytokinemia leading to multiorgan failure. In persisting liver inflammation, dominant profibrotic cytokines related to macrophages and stellate cells may enhance liver fibrosis. As gut-derived endotoxemia is considered to induce cytokine derangements and serious complications, proper managements of gut–liver axis are mandatory in patients with advanced liver diseases." @default.
- W2592518628 created "2017-03-16" @default.
- W2592518628 creator A5031685667 @default.
- W2592518628 date "2017-01-01" @default.
- W2592518628 modified "2023-09-25" @default.
- W2592518628 title "Cytokines in Hepatic Injury" @default.
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