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- W2593515375 abstract "Following advances in critical care, in-hospital multiple organ failure-related mortality is declining. Consequently, incidence of chronic critical illness is increasing. These patients linger in the intensive care unit, have high resource utilization, and poor long-term outcomes. Within this population, the authors propose that a substantial subset of patients have a new phenotype: persistent inflammation, immunosuppression, and catabolism syndrome. There is evidence that myelodysplasia with expansion of myeloid-derived suppressor cells, innate and adaptive immune suppression, and protein catabolism with malnutrition are major contributors. Optimal care of these patients will require novel multimodality interventions." @default.
- W2593515375 created "2017-03-16" @default.
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- W2593515375 date "2017-04-01" @default.
- W2593515375 modified "2023-10-18" @default.
- W2593515375 title "Persistent Inflammation, Immunosuppression and Catabolism Syndrome." @default.
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- W2593515375 doi "https://doi.org/10.1016/j.ccc.2016.12.001" @default.
- W2593515375 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/5351769" @default.
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