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- W2602428219 abstract "As the most mutated gene in cancer, it is no surprise that TP53 has been the center of cancer biology discourse since its discovery in the late 1970s. Although early demonstrations of p53's role in the modulation of cell proliferation and survival solidified its classification as a tumor suppressor and transcription factor, our conceptualization of p53 is ever-evolving. Here, we present novel evidence of the role of alternative splicing isoforms, truncating/separation-of-function mutations, and hotspot silent mutations in the regulation of p53's activities." @default.
- W2602428219 created "2017-04-07" @default.
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- W2602428219 date "2016-01-01" @default.
- W2602428219 modified "2023-09-27" @default.
- W2602428219 title "The Evolution of TP53 Mutations: From Loss-of-Function to Separation-of-Function Mutants." @default.
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- W2602428219 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/5298884" @default.
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