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• W2604032389 abstract "If no fertilization occurs at an appropriate time after ovulation, oocyte quality deteriorates rapidly as a process called postovulatory aging. Because the postovulatory aging of oocytes has detrimental effects on embryo development and offspring, many efforts have been made to prevent oocyte aging. Here we showed that quercetin prevented the decline in oocyte quality during postovulatory aging of oocytes. Quercetin treatment reduced aging-induced morphological changes and reactive oxygen species accumulation. Moreover, quercetin attenuated the aging-associated abnormalities in spindle organization and mitochondrial distribution, preventing decrease of SIRT expression and histone methylation. Quercetin also ameliorated the decrease in maturation-promoting factor activity and the onset of apoptosis during postovulatory aging. Furthermore, quercetin treatment during postovulatory aging improves early embryo development. Our results demonstrate that quercetin relieves deterioration in oocyte quality and improves subsequent embryo development." @default.
• W2604032389 created "2017-04-07" @default.
• W2604032389 creator A5017198015 @default.
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• W2604032389 creator A5019682947 @default.
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• W2604032389 date "2017-03-15" @default.
• W2604032389 modified "2023-10-18" @default.
• W2604032389 title "Quercetin delays postovulatory aging of mouse oocytes by regulating SIRT expression and MPF activity" @default.
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• W2604032389 doi "https://doi.org/10.18632/oncotarget.16219" @default.
• W2604032389 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/5503559" @default.
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• W2604032389 hasPublicationYear "2017" @default.
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