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- W2605609103 startingPage "123" @default.
- W2605609103 abstract "Regarding to the pathogenesis of renal arteriolar hyalinosis in diabetic nephropathy, we reported that glomerular circulatoly disturbance and Dysorie's theory (Schürmann and McMahorn, 1933) play on important parts in it's occurrence. Because, we found that the renal arteriolar hyalinosis most commonly occurred at vas. afference, it's near part and intraglomerular branches of vas. afference. This fact strongly suggested that the arteriolar hyalinosis is caused by the hemodynamic alteration due to circuratoly disturbance which might be taken place abruptly at the peripheral portion of intraglomerural afferent branches. But the genesis of this circuratloy disturbance is still unknown. The purpose for making clear on it, we examined that correlation between the morphology of diabetic glomerular lesion and arteriolar lesion to some clinical factors, duration of diabetes, hypertension, condition of DM-controle, proteinuria, serum creatinine level, rate of PSP-15 min., rate of inuline and PAH-clearance on 65 samples of kidney were obtained by autopsy. Result: 1) Duration of diabetes mellitus correlated to glomerular and arteriolar lesion. 2) Hypertension more correlated to arteriolar lesion than glomerular lesion. 3) Correlation is not found between FBS-controle in admission to glomerular lesion and arteriolar lesion, but the observations on samples of Brittle's type and diabetic coma, we found that poor controle of blood sugar level have had influence on arteriolar lesion. 4) Proteinuria is apt to precede to glomerular lesion in light microscopic finding. 5) Rate of PSP-15 min. and inuline and PAH-clearance correlated to glomerular and arteriolar lesion, but more strongly correlated to later one." @default.
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- W2605609103 date "1977-01-01" @default.
- W2605609103 modified "2023-09-25" @default.
- W2605609103 title "Histpathological Study of Renal Arteriosclerosis in Human Diabetes" @default.
- W2605609103 doi "https://doi.org/10.5551/jat1973.5.2_123" @default.
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