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- W2605731438 abstract "SUMMARY Plasmaconcentrations ofimmunoreactive atrial natriuretic peptide (mean(SEM)) were measured in135patients admitted totwo coronary careunits with myocardial infarction, ischaemic chest pain, ornon-ischaemic chest pain. Concentrations were significantly higher in patient's withacutemyocardial infarction nottreated withsystemic thrombolysis (604(14-3) pg/ ml)thaninpatients withnon-ischaemic chest pain(21 -1(43)pg/ml). Patients withischaemic chest painhadintermediate values (393(7-1)pg/ml). Patients withacutemyocardial infarction treated withintravenous streptokinase hadnormal concentrations ofplasma atrial natriuretic peptide (20'2 (36)pg/mg), which were significantly lower thanthose inpatients withmyocardial infarction not givenstreptokinase. Thesechanges could notbeexplained byfactors suchas age,pre-existing hypertension, renal dysfunction, orcardiac failure, nor treatmentother thanstreptokinase. Raised plasmaconcentrations ofatrial natriuretic peptide inacutemyocardial infarction may be a homoeostatic responseacting toreduce atrial pressuresbynatriuresis, diuresis, andvenodilatation. Thelowerconcentrations ofatrial natriuretic peptide inpatients withacute myocardial infarction treated withstreptokinase may reflect a short term beneficial haemodynamic effect of streptokinase. Plasmaconcentrations ofatrial natriuretic peptide areincreased byplasma volume expansion'-' andare higher thannormalinpatients withcongestive cardiac failure.° Thisincrease isprobably a response toatrial distension andisprobably a homoeostatic mechanism toloweratrial pressures through natriuresis, diuresis, andvenodilatation.4' Moststudies ofplasmaatrial natriuretic peptide concentrations inhumanventricular dysfunction havebeenperformed inpatients withchronic heart disease andovertfluid retention. We measured plasma concentrations ofatrial natriuretic peptide in patients without apast history ofcardiac failure when infarction wasessentially complete butbefore appreciable volumeexpansion couldoccur. We wanted tofind outwhether plasma concentrations of atrial natriuretic peptide rise inresponse toacute left ventricular muscledysfunction withoutvolume expansion. We alsoaimedtoassess theeffect on Requests forreprints toDrPA Phillips, Department ofMedicine, University ofMelbourne, AustinHospital, Heidelberg 3084, Victoria, Australia." @default.
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- W2605731438 date "2017-01-01" @default.
- W2605731438 modified "2023-09-27" @default.
- W2605731438 title "Plasma atrial natriuretic peptide inpatients with acutemyocardial infarction: effects ofstreptokinase" @default.
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