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- W2606571264 endingPage "21" @default.
- W2606571264 startingPage "14" @default.
- W2606571264 abstract "Aberrant innate immune system activation in the mother contributes greatly to the development of hypertension during pregnancy. Numerous groups have elicited vascular inflammation, endothelial dysfunction, and hypertension in animals during gestation by directly activating Toll-like receptors. Additionally, several experimental therapies that reduce pro-inflammatory immune cells and cytokines restore vascular endothelial function and normalize blood pressure. This review will summarize the research demonstrating that an excessive maternal innate immune response is sufficient to cause vascular inflammation and endothelial dysfunction, which contributes to the development of hypertension during pregnancy. Dampening the vascular inflammation caused by immune responses may reduce the incidence and severity of hypertensive disorders of pregnancy." @default.
- W2606571264 created "2017-04-28" @default.
- W2606571264 creator A5010517588 @default.
- W2606571264 creator A5018474833 @default.
- W2606571264 creator A5043369948 @default.
- W2606571264 creator A5062759098 @default.
- W2606571264 date "2017-07-01" @default.
- W2606571264 modified "2023-10-15" @default.
- W2606571264 title "Toll-like receptor activation, vascular endothelial function, and hypertensive disorders of pregnancy" @default.
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