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- W2608191330 endingPage "205" @default.
- W2608191330 startingPage "177" @default.
- W2608191330 abstract "Senescent cells arise as a consequence of cellular damage and can have either a detrimental or advantageous impact on tissues and organs depending on the specific cell type and metabolic state. As senescent cells accumulate in tissues with advancing age, they have been implicated in many age-related declines and diseases. The major facets of senescence include two pathways responsible for establishing and maintaining a senescence program, p53/CDKN1A(p21) and CDKN2A(p16)/RB, as well as the senescence-associated secretory phenotype. Numerous MicroRNAs influence senescence by modulating the abundance of key senescence regulatory proteins, generally by lowering the stability and/or translation of mRNAs that encode such factors. Accordingly, understanding the molecular mechanisms by which MicroRNAs influence senescence will enable diagnostic and therapeutic opportunities directed at senescent cells. Here, we review senescence-associated (SA)-MicroRNAs and discuss their implications in senescence-relevant pathologies." @default.
- W2608191330 created "2017-05-05" @default.
- W2608191330 creator A5006877808 @default.
- W2608191330 creator A5022033169 @default.
- W2608191330 creator A5032188964 @default.
- W2608191330 creator A5044404083 @default.
- W2608191330 creator A5064301050 @default.
- W2608191330 date "2017-01-01" @default.
- W2608191330 modified "2023-10-17" @default.
- W2608191330 title "Senescence-Associated MicroRNAs" @default.
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