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- W2613617081 abstract "Recently, it has been shown that proinflammatory cytokines play a complex and important role in the pathogenesis of many neurological disorders, including amyotrophic lateral sclerosis (ALS). To help facilitate future discoveries and more effective treatment strategies, we highlight the role that both innate and adaptive immune systems play in ALS and summarize the main observations that relate to cytokine expression levels in this disease. Furthermore, we propose a mechanism by which a known neurotoxin, β-N-methylamino-l-alanine (BMAA), may trigger this cytokine expression profile through motor neuron protein misfolding and subsequent NLRP3 (nucleotide-binding domain (NOD)-like receptor protein 3) inflammasome activation." @default.
- W2613617081 created "2017-05-19" @default.
- W2613617081 creator A5020480973 @default.
- W2613617081 creator A5064233348 @default.
- W2613617081 creator A5065806824 @default.
- W2613617081 creator A5071766929 @default.
- W2613617081 date "2017-10-01" @default.
- W2613617081 modified "2023-09-26" @default.
- W2613617081 title "Cytokine expression levels in ALS: A potential link between inflammation and BMAA-triggered protein misfolding" @default.
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- W2613617081 doi "https://doi.org/10.1016/j.cytogfr.2017.05.001" @default.
- W2613617081 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/28532674" @default.
- W2613617081 hasPublicationYear "2017" @default.
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