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- W2625418525 endingPage "184" @default.
- W2625418525 startingPage "175" @default.
- W2625418525 abstract "Summary Altered metabolism is a hallmark of cancers, including shifting oxidative phosphorylation to glycolysis and up‐regulating glutaminolysis to divert carbon sources into biosynthetic pathways that promote proliferation and survival. Therefore, metabolic inhibitors represent promising anti‐cancer drugs. However, T cells must rapidly divide and survive in harsh microenvironments to mediate anti‐cancer effects. Metabolic profiles of cancer cells and activated T lymphocytes are similar, raising the risk of metabolic inhibitors impairing the immune system. Immune checkpoint blockade provides an example of how metabolism can be differentially impacted to impair cancer cells but support T cells. Implications for research with metabolic inhibitors are discussed." @default.
- W2625418525 created "2017-06-23" @default.
- W2625418525 creator A5000013720 @default.
- W2625418525 creator A5046228493 @default.
- W2625418525 creator A5077171756 @default.
- W2625418525 date "2017-07-10" @default.
- W2625418525 modified "2023-10-16" @default.
- W2625418525 title "Metabolic reprogramming in the tumour microenvironment: a hallmark shared by cancer cells and T lymphocytes" @default.
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