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- W2625934676 endingPage "1389" @default.
- W2625934676 startingPage "1373" @default.
- W2625934676 abstract "Abstract Wild mammals have a diversity of reproductive strategies and endocrine adaptations to survive and reproduce under hostile environmental conditions. The endocrine system has highly integrated cause and effect physiology with genetic and epigenetic interactivity during a life history. Embryo fetal development is an once-in-a-lifetime event. The fetus has inherent plasticity under maternal influences, and anthropogenic chemicals can have toxicophysiological effects on fetal plasticity. Endocrine-disrupting chemicals (EDCs) in the fetus can cause congenital misprogramming of life stages of the endocrine system, and expression may not occur until puberty and adult life. Congenital changes to the endocrine system also can cause unexpected sensitivity to environmental stimuli. Exposure to EDCs can alter reproductive outcomes. Female fecundity is generally considered to be more sensitive in terms of environmental levels of EDCs required to lower the total lifetime progeny as compared with the environmental level of EDCs that would decrease the fecundity of males. The environmental chemistry of persistent organic pollutants (POPs) is complex, and some POPs may become more toxic after being released into the environment. Exposure of wild mammals to EDCs can occur in utero, from milk ingested during nursing and from the food web. There is also interactivity of the different pollutants in fauna at various life stages. Physical agents can alter reproductive successes." @default.
- W2625934676 created "2017-06-23" @default.
- W2625934676 creator A5042622656 @default.
- W2625934676 creator A5055385690 @default.
- W2625934676 date "2017-01-01" @default.
- W2625934676 modified "2023-10-01" @default.
- W2625934676 title "Endocrine Disruption in Wildlife Species" @default.
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