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- W2626921567 abstract "Abstract : Malaria, a disease caused by an intracellular parasite of the genus Plasmodium, causes 350-500 million cases annually with 1-2 million deaths. The majority of these deaths occur in children in sub-Saharan Africa due to complications of P. falciparum infection, such as severe malarial anemia (SMA). The pathogenesis of SMA is complex and not well understood; however, it is known that there is greater destruction of red cells than can be accounted for by the parasite alone. The question of what is happening to the uninfected red cells remains unanswered. We therefore investigated the pathogenesis of SMA by studying the fate of uninfected red cells in two different mouse models. Based on findings in patients with SMA showing an acquired deficiency in complement regulatory proteins and increased susceptibility to complement, we emphasized investigating the role of complement and complement regulatory proteins in the development of SMA." @default.
- W2626921567 created "2017-06-23" @default.
- W2626921567 creator A5082526975 @default.
- W2626921567 date "2011-02-07" @default.
- W2626921567 modified "2023-09-23" @default.
- W2626921567 title "The Fate of the Red Cells: Insights from Two Models of Severe Malarial Anemia" @default.
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- W2626921567 doi "https://doi.org/10.21236/ad1013332" @default.
- W2626921567 hasPublicationYear "2011" @default.
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