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• W2671569358 abstract "We investigated the skeletal muscle adaptation to l ‐arginine supplementation prior to a single session of resistance exercise ( RE ) during the early phase of muscle repair. Wistar rats were randomly assigned into non‐exercised (Control), RE plus vehicle ( RE ); RE plus l ‐arginine ( RE +L‐arg) and RE plus aminoguanidine ( RE + AG ) groups. Animals received four doses of either vehicle (0.9% NaCl), l ‐arg (1 g/b.w.), or AG ( iNOS inhibitor) (50 mg/b.w.). The animals performed a single RE session until the concentric failure (ladder climbing; 80% overload) and the skeletal muscles were harvested at 0, 8, 24, and 48 hours post‐ RE . The RE resulted in increased neutrophil infiltrate (24 hours post‐ RE ) (3621 vs 11852; P <.0001) associated with enhanced TNF ‐α (819.49 vs 357.02; P <.005) and IL ‐6 (3.84 vs 1.08; P <.0001). Prior, l ‐arginine supplementation attenuates neutrophil infiltration (5622; P <.0001), and also TNF ‐α (506.01; P <.05) and IL ‐6 (2.51, P <.05) levels. AG pretreatment mediated an inhibition of iNOS levels similar to levels found in RE group. RE animals displayed increased of atrogin‐1 (1.9 fold) and Mu RF ‐1 (3.2 fold) mRNA levels, reversed by l ‐arg supplementation [atrogin‐1 (0.6 fold; P <.001); Mu RF ‐1 (0.8‐fold; P <.001)] at 24 hours post‐ RE . MyoD up‐regulated levels were restricted to l ‐arg treated animals at 24 hours (2.8 vs 1.5 fold; P <.005) and 48 hours post‐ RE (2.4 vs 1.1 fold; P <.001). AG pretreatment reversed these processes at 24 hours [atrogin‐1 (2.1 fold; P <.0001); Mu RF ‐1 (2.5 fold; P <.0001); MyoD (1.4 fold)]. l ‐arginine supplementation seems to attenuate the resolution of RE ‐induced muscle inflammation and up‐regulates MyoD expression during the early phase of muscle repair." @default.
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• W2671569358 date "2017-08-04" @default.
• W2671569358 modified "2023-10-03" @default.
• W2671569358 title "<scp>l</scp>-arginine modulates inflammation and muscle regulatory genes after a single session of resistance exercise in rats" @default.
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• W2671569358 doi "https://doi.org/10.1111/sms.12935" @default.
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