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- W2685066188 abstract "Spontaneous mutations in mice, along with those generated by genetic engineering approaches, are essential tools to understand the pathogenesis of human epilepsies. Monogenic models provide a coherent, reproducible lesion and a tractable biological starting point to explore the remarkably diverse biology of seizure disorders in the developing brain. Current clinical classification schemes estimate that about two thirds of all human epilepsies, formerly deemed idiopathic, are now considered to be of primarily genetic origin. Over 150 monogenic epilepsies have been identified. For each of these inherited disorders, a genetically defined mouse mutant can help identify the key molecular defects in excitability and plasticity in the developing epileptic brain, uncover new molecular targets for therapy, and serve as a reproducible biological test system for experimental strategies to prevent or reverse the onset of seizures." @default.
- W2685066188 created "2017-06-30" @default.
- W2685066188 creator A5012194422 @default.
- W2685066188 date "2017-01-01" @default.
- W2685066188 modified "2023-09-27" @default.
- W2685066188 title "Spontaneous and Gene-Directed Epilepsy Mutations in the Mouse" @default.
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- W2685066188 doi "https://doi.org/10.1016/b978-0-12-804066-9.00052-3" @default.
- W2685066188 hasPublicationYear "2017" @default.
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