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• W2734183746 abstract "Abstract Acute kidney injury (AKI) represents a significant clinical concern that is associated with high mortality rates and also represents a significant risk factor for the development of chronic kidney disease (CKD). This article will consider alterations in renal endothelial function in the setting of AKI that may underlie impairment in renal perfusion and how inefficient vascular repair may manifest post‐AKI and contribute to the potential transition to CKD. We provide updated terminology for cells previously classified as ‘endothelial progenitor’ that may mediate vascular repair such as pro‐angiogenic cells and endothelial colony‐forming cells. We consider how endothelial repair may be mediated by these different cell types following vascular injury, particularly in models of AKI. We further summarize the potential ability of these different cells to mitigate the severity of AKI, improve perfusion and maintain vascular structure in pre‐clinical studies." @default.
• W2734183746 created "2017-07-14" @default.
• W2734183746 creator A5026654280 @default.
• W2734183746 creator A5051594076 @default.
• W2734183746 creator A5059657108 @default.
• W2734183746 date "2017-07-25" @default.
• W2734183746 modified "2023-10-13" @default.
• W2734183746 title "Endothelial colony-forming cells and pro-angiogenic cells: clarifying definitions and their potential role in mitigating acute kidney injury" @default.
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