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- W2743126337 endingPage "170" @default.
- W2743126337 startingPage "147" @default.
- W2743126337 abstract "Lung disease is a global health burden, contributing to significant morbidity and mortality. Programming of the lung following adverse environmental exposures during development contributes to the development of lung disease throughout the lifespan. A wide variety of adverse environmental exposures during prenatal and early postnatal development cause developmental programming of the lung. One well-studied exposure linked to the developmental programming of lung disease is maternal tobacco smoke and isolated nicotine. Maternal tobacco use contributes to programming of the fetal lung directly and via effects on the placenta. Mechanistically, epigenetic effects contribute to the programming of lung disease. Epigenetic mechanisms include DNA methylation, histone protein modifications, and noncoding RNAs. Studies support an association between perinatal insults and epigenetic modifications. As research directives address the programming of lung disease in human studies and in animal models, the goal is to develop targeted interventions that may be applied during vulnerable developmental windows, thus diverting the development of lung disease, and alleviate the associated global health burdens." @default.
- W2743126337 created "2017-08-17" @default.
- W2743126337 creator A5006784317 @default.
- W2743126337 creator A5029943512 @default.
- W2743126337 creator A5040989106 @default.
- W2743126337 date "2018-01-01" @default.
- W2743126337 modified "2023-09-27" @default.
- W2743126337 title "Environmental Exposures and Developmental Programming of the Lung" @default.
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