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- W2743596823 abstract "Gut microbes are key partners in host defense against potential pathogens ( 1 ). This might be achieved through cross-talk between gut bacteria, epithelial cells lining the gut (colonocytes), and immune cells ( 2 ). Part of this cross-talk involves metabolites derived from the bacteria, such as the short-chain fatty acid butyrate. This can bind to specific G protein-coupled receptors in colonocytes and immune cells, leading to antimicrobial immune responses ( 3 ). However, what if this cross-talk were not the full story? On page 570 of this issue, Byndloss et al. ( 4 ) show that butyrate instructs colonocytes to consume oxygen through the β-oxidation metabolic pathway and consequently protects the host against the expansion of potentially pathogenic bacteria that can lead to inflammatory bowel diseases." @default.
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- W2743596823 date "2017-08-10" @default.
- W2743596823 modified "2023-10-18" @default.
- W2743596823 title "Gut cell metabolism shapes the microbiome" @default.
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- W2743596823 doi "https://doi.org/10.1126/science.aao2202" @default.
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