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• W2747149289 abstract "Significance The therapeutic activity of checkpoint blockers and toll-like receptor (TLR) agonists, which show some efficacy against malignancies, appears to at least partially result from the secretion of type-I IFNs. Thus, we hypothesized that type-I IFN-inducible transcription factors, such as basic leucine zipper transcription factor ATF-like 2 ( Batf2 ), might play a role in tumor immunity. Here, we investigated the role of Batf2 , especially its positive transcriptional activities, and evaluated its antitumor effect. This study shows that Batf2 has an antitumor effect through the up-regulation of IL-12 p40 in tumor-associated macrophages, which eventually induces the activation of CD8 + T cells and their accumulation within the tumor. Batf2 may be an important target in anticancer treatment with immune checkpoint blockers and TLR agonists." @default.
• W2747149289 created "2017-08-31" @default.
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• W2747149289 date "2017-08-14" @default.
• W2747149289 modified "2023-10-16" @default.
• W2747149289 title "Antitumor effect of <i>Batf2</i> through IL-12 p40 up-regulation in tumor-associated macrophages" @default.
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• W2747149289 doi "https://doi.org/10.1073/pnas.1708598114" @default.
• W2747149289 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/5584452" @default.
• W2747149289 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/28808017" @default.
• W2747149289 hasPublicationYear "2017" @default.
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