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- W2753738234 abstract "Cancer is a complex disease where cancer stem cells (CSCs) maintain unlimited replicative potential, but evade chemotherapy drugs through cellular quiescence. CSCs are able to give rise to bulk tumor cells that have the capability to override antiproliferative signals and evade apoptosis. Numerous pathways are dysregulated in tumor cells, where increased levels of prooxidant reactive oxygen and nitrogen species can lead to localized inflammation to exacerbate all three stages of tumorigenesis: initiation, progression, and metastasis. Modulation of cellular metabolism in tumor cells as well as immune cells in the tumor microenvironment (TME) can impact inflammatory networks. Altering these pathways can potentially serve as a portal for therapy. It is well known that selenium, through selenoproteins, modulates inflammatory pathways in addition to regulating redox homeostasis in cells. Therefore, selenium has the potential to impact the interaction between tumor cells, CSCs, and immune cells. In the sections later, we review the current status of knowledge regarding this interaction, with reference to leukemia stem cells, and the importance of selenium-dependent regulation of inflammation as a potential mechanism to affect the TME and tumor cell survival." @default.
- W2753738234 created "2017-09-15" @default.
- W2753738234 creator A5005427609 @default.
- W2753738234 creator A5016440386 @default.
- W2753738234 creator A5044845960 @default.
- W2753738234 creator A5082882581 @default.
- W2753738234 date "2017-01-01" @default.
- W2753738234 modified "2023-10-16" @default.
- W2753738234 title "The Regulation of Pathways of Inflammation and Resolution in Immune Cells and Cancer Stem Cells by Selenium" @default.
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