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- W2754281752 endingPage "181" @default.
- W2754281752 startingPage "159" @default.
- W2754281752 abstract "Neurotoxicity due to excessive exposure to manganese (Mn) has been described as early as 1837 (Couper, Br Ann Med Pharm Vital Stat Gen Sci 1:41–42, 1837). Extensive research over the past two decades has revealed that Mn-induced neurological injury involves complex pathophysiological signaling mechanisms between neurons and glial cells. Glial cells are an important target of Mn in the brain, both for sequestration of the metal, as well as for activating inflammatory signaling pathways that damage neurons through overproduction of numerous reactive oxygen and nitrogen species and inflammatory cytokines. Understanding how these pathways are regulated in glial cells during Mn exposure is critical to determining the mechanisms underlying permanent neurological dysfunction stemming from excess exposure. The subject of this review will be to delineate mechanisms by which Mn interacts with glial cells to perturb neuronal function, with a particular emphasis on neuroinflammation and neuroinflammatory signaling between distinct populations of glial cells." @default.
- W2754281752 created "2017-09-25" @default.
- W2754281752 creator A5002274261 @default.
- W2754281752 creator A5066895643 @default.
- W2754281752 creator A5086123386 @default.
- W2754281752 date "2017-01-01" @default.
- W2754281752 modified "2023-10-18" @default.
- W2754281752 title "Inflammatory Activation of Microglia and Astrocytes in Manganese Neurotoxicity" @default.
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