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• W2756297428 abstract "(Cancer Cell 16, 533–546; December 8, 2009) In this article, the affiliation of the coauthor Piergiorgio Modena was listed incorrectly as Istituto Nazionale per lo Studio e la Cura dei Tumori, Milano, 210133, Italy, and the postal codes of Hospital for Sick Children and Ontario Institute of Cancer Research were mistakenly reversed. In addition, M. Massimino was incorrectly listed as M. Massimo in the Acknowledgments. The correct author affiliations and Acknowledgments are printed below: 1Division of Hematology-Oncology 2Division of Neurosurgery 3Department of Pathology 4The Center for Applied Genomics Hospital for Sick Children, Toronto, ON M5G 1X8, Canada 5Ontario Institute of Cancer Research, Toronto, ON M5G 0A3, Canada 6Division of Pathology, John Hopkins University School of Medicine, Baltimore, MD 21205, USA 7Division of Pathology, University of Cambridge, Cambridge CB2ITN, UK 8Department of Neurosurgery, Virginia Commonwealth University, Richmond, VA 23298-063, USA 9Unit of Experimental Oncology 1 CRO, Aviano National Cancer Institute Via F. Gallini 2, AVIANO 33081 (PN) Italy 10Department of Neurosurgery, Taiwan Veteran's General Hospital, Taiwan, ROC 11Department of Neurosurgery, Nanfang Hospital, Guangzhou, 510515, China 12Department of Neurology, Children's Hospital Boston, Boston, MA 02115, USA 13Texas Children's Cancer Center, Baylor College of Medicine, Houston, TX 77030, USA 14Neuro-oncology Division, St. Jude Children's Research Hospital; Memphis, TN 38105-3678, USA We thank P. Burger, J. Squire, L. Penn for mentorship; M. Massimino, L. Lafay-Cousin, M. Zielenska, S. Chilton, P. Northcott, P. Kongkam, T. Paton and G. Casallo for generous help; J. Dick, S. Egan, C.C. Hui, E. Lechman, R. Gilbertson, W. Halliday for helpful discussions; P. Hu for statistical analysis. Funding from CBTF, SickKids (Brainchild), JM Foundations and CCSRI (grant #17473) to A.H., and AIRC to P.M. and tissue contributions from CHTN, NINDs and the London Regional Brain Tumor Bank are gratefully acknowledged. M.L. was an ABTA Fellow; K.F.L. and D.S. received SickKids foundation RESTRACOMP predoctoral awards. M.L., K.F.L., Y.L. and D.S. performed all experiments and analyses with support from D.P., L.Z. and P.B. NSC studies were performed by I.C.; E.B., P.M., M.L., P.C., M.C., T.V., S.P., C.E., and C.L. provided tumor tissue and clinical data. A.H. supervised the project. A.H., M.D.T., S.S., J.T.R., C.E.H., and P.B.D. contributed to manuscript writing and review. Frequent Amplification of a chr19q13.41 MicroRNA Polycistron in Aggressive Primitive Neuroectodermal Brain TumorsLi et al.Cancer CellDecember 08, 2009In BriefWe discovered a high-level amplicon involving the chr19q13.41 microRNA (miRNA) cluster (C19MC) in 11/45 (∼25%) primary CNS-PNET, which results in striking overexpression of miR-517c and 520g. Constitutive expression of miR-517c or 520g promotes in vitro and in vivo oncogenicity, modulates cell survival, and robustly enhances growth of untransformed human neural stem cells (hNSCs) in part by upregulating WNT pathway signaling and restricting differentiation of hNSCs. Remarkably, the C19MC amplicon, which is very rare in other brain tumors (1/263), identifies an aggressive subgroup of CNS-PNET with distinct gene-expression profiles, characteristic histology, and dismal survival. Full-Text PDF Open Archive" @default.
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• W2756297428 title "Frequent Amplification of a chr19q13.41 MicroRNA Polycistron in Aggressive Primitive Neuroectodermal Brain Tumors" @default.
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