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- W2765791353 startingPage "258" @default.
- W2765791353 abstract "In cancer progression, metastasis is a major cause of poor survival of patients and can be targeted for therapeutic interventions. The first discovered metastatic-suppressor Nm23-H1 possesses nucleoside diphosphate kinase, histidine kinase, and DNase activity as a broad-spectrum enzyme. Recent advances in cancer metastasis have opened new ways for the development of therapeutic molecular approaches. In this review, we provide a summary of the current understanding of Nm23/NDPKs in the context of viral oncogenesis. We also focused on Nm23-H1-mediated cellular events with an emphasis on chromatin modifications. How Nm23-H1 modulates the activities of chromatin modifiers through interaction with Epstein-Barr virus-encoded oncogenic antigens and related crosstalks are discussed in the context of other oncogenic viruses. We also described the current understanding of the cellular and viral interactions of Nm23-H1 and their reference to transcription regulation and metastasis. Further, we summarized the recent therapeutic approaches targeting Nm23 and its potential links to pathways that can be exploited by oncogenic viruses." @default.
- W2765791353 created "2017-11-10" @default.
- W2765791353 creator A5032066345 @default.
- W2765791353 creator A5034148348 @default.
- W2765791353 date "2018-02-01" @default.
- W2765791353 modified "2023-10-18" @default.
- W2765791353 title "Oncogenic Epstein–Barr virus recruits Nm23-H1 to regulate chromatin modifiers" @default.
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