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• W2766005170 abstract "Heart failure (HF) continues to be a public health burden despite advances in therapy, and the natriuretic peptide (NP) system is clearly of critical importance in this setting, spawning valuable diagnostic and prognostic testing, such as B-type natriuretic peptide (BNP) and N-terminal pro-BNP (NT-proBNP), as well as current and future therapeutics, including recombinant natriuretic peptides (e.g., carperitide, nesiritide) and recently sacubitril, which inhibits the key clearance mechanism for NPs. This article intends to summarize the existing evidence for the role of NP system genetic variation on cardiovascular phenotypes relevant to HF with particular focus on the potential impact on pharmacologic therapies.Several genes in NP system have been interrogated, in many cases genetic variation impacting protein quantity and function or related disease states. Recent data supports genetic variants potentially impacting pharmacokinetics or dynamics of medications targeting the pathway. Growing evidence indicates the importance of genetic variation to the functioning of the NP system and its pharmacologic manipulation." @default.
• W2766005170 created "2017-11-10" @default.
• W2766005170 creator A5028502868 @default.
• W2766005170 creator A5085969872 @default.
• W2766005170 date "2017-10-27" @default.
• W2766005170 modified "2023-10-15" @default.
• W2766005170 title "Pharmacogenomics of the Natriuretic Peptide System in Heart Failure" @default.
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• W2766005170 doi "https://doi.org/10.1007/s11897-017-0365-5" @default.
• W2766005170 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/5681877" @default.
• W2766005170 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/29075957" @default.
• W2766005170 hasPublicationYear "2017" @default.
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