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- W2766277098 endingPage "50" @default.
- W2766277098 startingPage "29" @default.
- W2766277098 abstract "Abstract The cognitive deficits in patients with Alzheimer’s disease (AD) are associated with synaptic dysfunction. In early stages accumulation of Abeta oligomers might damage the synapses while in later stages tau and neurofibrillary pathology will play a key role. It is apparent from this sequence of events that cognitive decline is likely to occur in someone with AD well before the clinical diagnosis can be made with any certainty. Age-related cognitive decline is evident in information processing abilities such as effortful encoding of new information, processing speed, inductive reasoning, and working memory. In AD there is evidence that episodic memory deteriorates rapidly. Identification of the initial cognitive changes of AD can help to reliably detect the disease in its earliest stages and provide a target for treatment. The combined use of neuropsychological assessment and biomarkers of AD may be particularly effective." @default.
- W2766277098 created "2017-11-10" @default.
- W2766277098 creator A5074775445 @default.
- W2766277098 creator A5081258864 @default.
- W2766277098 date "2016-01-01" @default.
- W2766277098 modified "2023-09-26" @default.
- W2766277098 title "When Cognitive Decline Becomes Pathology" @default.
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