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- W2766337005 abstract "Some clinical subtypes of dementias with posterior lobar degeneration have been described as atypical Alzheimer's disease (AD). Patients with posterior cortical atrophy (PCA), logopenic progressive aphasia (LPA), or primary progressive apraxia frequently have AD pathologies. However, not every atypical AD patient fall within the range of these clinical subtypes. Case report with neuropsychological and neuroimaging data. A 73 years old, right handed female with 12 years of education, having no previous medical or psychiatric problems, began to experience progressive agraphia of Kanji (Japanese morphograms), while she retained good reading Kanji and good reading and writing Kana (Japanese syllabograms). She was no longer able to write her husband's name in kanji, so that she visited our memory clinic for evaluation of writing difficulties one year after the onset of agraphia. She had been housewife and enjoyed volunteer job in storytelling without any problems. The Standard Language Test of Aphasia indicated that she was normal in articulation, fluent in speaking, capable in language comprehension and repetition. Her memory deficit was not apparent (the scores on logical memory subtest of the Wechsler Memory Scale were 7 in immediate recall and 5 in delayed recall). She performed slightly worse on the Rey-Osterrieth complex figure test (copy and delayed recall) and the Raven's colored progressive matrices test, which might indicate her visuospatial dysfunction. Given positive findings of amyloid PET using florbetaben (18F), she was concluded as a prodromal AD. While having normal findings in brain MRI, IMP-SPECT demonstrated relative hypoperfusion mainly in the left parietal areas which partly extended to the temporal lobe. These affected areas were slightly posterior from the perisylvian area (which usually affected by LPA). Also, posterior lobe areas were not affected on this case. Our case report might suggest the existence of a subgroup of atypical AD, presenting posterior symptoms such as agraphia, acalculia and visuospatial deficits, without showing the signs of aphasia or agnosia. This subgroup may reflect the spectrum of clinical manifestations between LPA and PCA." @default.
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- W2766337005 date "2017-07-01" @default.
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- W2766337005 title "AGRAPHIA AS AN INITIAL SYMPTOM OF ALZHEIMER'S DISEASE: CASE REPORT" @default.
- W2766337005 doi "https://doi.org/10.1016/j.jalz.2017.07.641" @default.
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