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- W2767764306 abstract "Rapid developments in free radical biology and molecular technology have permitted exploration of the free radical theory of aging. Oxidative stress has also been implicated in the pathogenesis of a number of diseases. Studies have found evidence of oxidative damage to macromolecules (DNA, lipids, protein), and data in transgenic Drosophila melanogaster support the hypothesis that oxidative injury might directly cause the aging process. Additional links between oxidative stress and aging focus on mitochondria, leading to development of the mitochondrial theory of aging. However, despite the number of studies describing the association of markers of oxidative damage with advancing age, few, if any definitively link oxidative injury to altered energy production or cellular function. Although a causal role for oxidative stress in the aging process has not been clearly established, this does not preclude attempts to reduce oxidative injury as a means to reduce morbidity and perhaps increase the healthy, usefu..." @default.
- W2767764306 created "2017-11-17" @default.
- W2767764306 creator A5011859104 @default.
- W2767764306 date "2016-11-29" @default.
- W2767764306 modified "2023-09-23" @default.
- W2767764306 title "Aging: Is Oxidative Stress a Marker or is it Causal? (44454)" @default.
- W2767764306 doi "https://doi.org/10.1177/153537029922200312" @default.
- W2767764306 hasPublicationYear "2016" @default.
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