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- W2772260227 abstract "Abstract Early atherosclerosis features functional and structural changes in the endothelial barrier function that affect the traffic of molecules and solutes between the vessel lumen and the vascular wall. Such changes are mechanistically related to the development of atherosclerosis. Proatherogenic stimuli and cardiovascular risk factors, such as dyslipidaemias, diabetes, obesity, and smoking, all increase endothelial permeability sharing a common signalling denominator: an imbalance in the production/disposal of reactive oxygen species (ROS), broadly termed oxidative stress. Mostly as a consequence of the activation of enzymatic systems leading to ROS overproduction, proatherogenic factors lead to a pro-inflammatory status that translates in changes in gene expression and functional rearrangements, including changes in the transendothelial transport of molecules, leading to the deposition of low-density lipoproteins (LDL) and the subsequent infiltration of circulating leucocytes in the intima. In this review, we focus on such early changes in atherogenesis and on the concept that proatherogenic stimuli and risk factors for cardiovascular disease, by altering the endothelial barrier properties, co-ordinately trigger the accumulation of LDL in the intima and ultimately plaque formation." @default.
- W2772260227 created "2017-12-22" @default.
- W2772260227 creator A5012146183 @default.
- W2772260227 creator A5035619683 @default.
- W2772260227 creator A5036907767 @default.
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- W2772260227 creator A5067302152 @default.
- W2772260227 creator A5081233977 @default.
- W2772260227 creator A5083354719 @default.
- W2772260227 date "2017-12-08" @default.
- W2772260227 modified "2023-10-17" @default.
- W2772260227 title "Endothelial permeability, LDL deposition, and cardiovascular risk factors—a review" @default.
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