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- W2775287789 endingPage "596" @default.
- W2775287789 startingPage "575" @default.
- W2775287789 abstract "Insulin resistance, a common feature of metabolic disorders such as obesity, nonalcoholic fatty liver disease, metabolic syndrome, and polycystic ovary syndrome, is a risk factor for development of diabetes. Because sex hormones orchestrate the establishment of sex-specific behavioral, reproductive, and metabolic differences, a role for them in the developmental origin of insulin resistance is also to be expected. Female sheep exposed to male levels of testosterone during fetal life serve as an excellent translational model for delineating programming of insulin resistance. This chapter summarizes the ontogeny of insulin resistance, the tissue-specific changes in insulin sensitivity, and the various factors that are involved in the programming and maintenance of the insulin resistance in adult female sheep that were developmentally exposed to fetal male levels of testosterone during the sexual-differentiation window." @default.
- W2775287789 created "2017-12-22" @default.
- W2775287789 creator A5029119143 @default.
- W2775287789 creator A5053708510 @default.
- W2775287789 date "2017-01-01" @default.
- W2775287789 modified "2023-09-24" @default.
- W2775287789 title "Prenatal Testosterone Programming of Insulin Resistance in the Female Sheep" @default.
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- W2775287789 doi "https://doi.org/10.1007/978-3-319-70178-3_25" @default.
- W2775287789 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/29224111" @default.
- W2775287789 hasPublicationYear "2017" @default.
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