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• W2777309068 abstract "Highlights •The reactive oxygen species–generating enzyme NADPH oxidase 4 (Nox4) is up-regulated in the heart after myocardial infarction. •Mice with cardiomyocyte-targeted overexpression of Nox4 display an increase in macrophages in the heart, with skewing of polarization toward an M2 phenotype. •After myocardial infarction, Nox4-induced skewing of macrophage polarization toward an M2 phenotype is accompanied by a higher survival, decreased cardiac remodeling, and improved contractile function. •The protective effects of cardiomyocyte Nox4 may, in part, involve the attenuation of cardiac matrix metalloproteinase–2 activity." @default.
• W2777309068 created "2018-01-05" @default.
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• W2777309068 date "2017-12-01" @default.
• W2777309068 modified "2023-10-03" @default.
• W2777309068 title "NADPH Oxidase-4 Driven Cardiac Macrophage Polarization Protects Against Myocardial Infarction–Induced Remodeling" @default.
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• W2777309068 doi "https://doi.org/10.1016/j.jacbts.2017.06.006" @default.
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