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• W2777492072 abstract "Over the past decade, evidence has emerged suggesting a broader role for cytochrome c (Cyt c ) in programmed cell death. Recently, we demonstrated the ability of Cyt c to inhibit the nucleosome assembly activity of histone chaperones SET/template‐activating factor Iβ and NAP1‐related protein during DNA damage in humans and plants respectively. Here, we hypothesise a dual concentration‐dependent function for nuclear Cyt c in response to DNA damage. We propose that low levels of highly cytotoxic DNA lesions – such as double‐strand breaks – induce nuclear translocation of Cyt c , leading to the attenuation of nucleosome assembly and, thereby, increasing the time available for DNA repair. If DNA damage persists or is exacerbated, the nuclear Cyt c concentration would exceed a given threshold, causing the haem protein to block DNA remodelling altogether." @default.
• W2777492072 created "2018-01-05" @default.
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• W2777492072 date "2018-01-01" @default.
• W2777492072 modified "2023-10-18" @default.
• W2777492072 title "Nuclear cytochrome <i>c</i> – a mitochondrial visitor regulating damaged chromatin dynamics" @default.
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• W2777492072 doi "https://doi.org/10.1002/1873-3468.12959" @default.
• W2777492072 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/29288494" @default.
• W2777492072 hasPublicationYear "2018" @default.
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