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- W2778902474 endingPage "5683" @default.
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- W2778902474 abstract "The knowledge of the cellular events occurring in the aging heart has dramatically expanded in the last decade and is expected to further grow in years to come. It is now clear that impaired function and loss of cardiomyocytes are major features of cardiac aging, but other events are likewise important. In particular, accumulating experimental evidence highlights the importance of fibroblast and cardiac progenitor cell (CPC) dysfunction. The Notch pathway regulates cardiomyocyte, fibroblast, and CPC activity and, thus, may be critically involved in heart disease associated with advanced age, especially heart failure. In a translational perspective, thorough investigation of the Notch system in the aging myocardium may lead to the identification of molecular targets for novel therapies for age-related cardiac disease." @default.
- W2778902474 created "2018-01-05" @default.
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- W2778902474 creator A5037942689 @default.
- W2778902474 creator A5047109750 @default.
- W2778902474 creator A5062655570 @default.
- W2778902474 date "2018-02-28" @default.
- W2778902474 modified "2023-09-24" @default.
- W2778902474 title "Beyond cardiomyocyte loss: Role of Notch in cardiac aging" @default.
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