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- W277927358 abstract "Develop-ment-mechanism of chronic glomerulonephritis (the contracted kidney) was discussed in this paper, on the basis of both clinical observations which has recently been accumulated especially from comparative studies on (a) vascular and glomerular changes of chronic glomerulonephritis, lupus nephritis, diabetic nephropathy and so on and (b) a new experimental animal model of adult human chronic glomerulonephritis, which is induced in homologous animals by a single footpads injection of nephritogenoside.Nephritogenoside was isolated and purified from the glomerular basement membrane of normal and healthy rats. Nephritogenoside has no antigenic activity but has biologic activity that induces, chemically, chronic glomerulonephritis (the contracted kidney) . Chemical structure of the active portion of nephritogenoside (sugar portion) has already established as follows; Glc (α1→6) Glc (β1→6) Glc (α1→NHCO-peptide) (a synthesized compound (Glc (α1→6) Glc (β1→6) Glc (α1→NHCO-Asn or Gln) showed the same 13CNMR specrum with the sugar portion of nephritogenoside)" @default.
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- W277927358 date "1983-01-01" @default.
- W277927358 modified "2023-10-18" @default.
- W277927358 title "On the Development-mechanism of chronic glomerulonephritis (the contracted kidney)" @default.
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- W277927358 doi "https://doi.org/10.2492/jsir1981.3.3_191" @default.
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