FRINK Linked Data Fragments server

Matches in SemOpenAlex for { <https://semopenalex.org/work/W2779893991> ?p ?o ?g. }

• W2779893991 endingPage "194" @default.
• W2779893991 startingPage "183" @default.
• W2779893991 abstract "Primary muscle tension dysphonia (pMTD) is a voice disorder that occurs in the absence of laryngeal pathology. Dysregulated activity of the paralaryngeal muscles is considered the proximal cause; however, the central origin of this aberrant laryngeal muscle activation is unclear. The Trait Theory (Roy and Bless, 2000a,b) proposed that specific personality traits can predispose one to laryngeal motor inhibition and pMTD, and this inhibition is mediated by a hyperactive “behavioral inhibition system (BIS)” composed of limbic system structures (and associated prefrontal connections). This case study used functional magnetic resonance imaging to detect brain activation changes associated with successful management of pMTD, thereby evaluating possible neural correlates of this poorly understood disorder. Method A 61-year-old woman with moderate-to-severe pMTD underwent functional magnetic resonance imaging scans before and immediately after successful treatment using manual circumlaryngeal techniques. Experimental stimuli were blocks of repeated vowel production and overt sentence reading. Results Significantly greater activation was observed pre- versus posttreatment in all regions of interest during sentence production, that is, periaqueductal gray, amygdala, hypothalamus, anterior cingulate cortex, hippocampus, dorsolateral prefrontal cortex, Brodmann area 10, and premotor and inferior sensorimotor cortex. Conclusions Our findings are compatible with overactivation of neural regions associated with the BIS (cingulate cortex, amygdala, hypothalamus, periaqueductal gray) and motor inhibition networks (eg, [pre-]supplementary motor area) along with the dorsolateral prefrontal cortex and medial prefrontal cortex. Heightened input from limbic regions combined with dysfunctional prefrontal regulation may interfere with laryngeal motor preparation, initiation, and execution thereby contributing to disordered voice in pMTD. Primary muscle tension dysphonia (pMTD) is a voice disorder that occurs in the absence of laryngeal pathology. Dysregulated activity of the paralaryngeal muscles is considered the proximal cause; however, the central origin of this aberrant laryngeal muscle activation is unclear. The Trait Theory (Roy and Bless, 2000a,b) proposed that specific personality traits can predispose one to laryngeal motor inhibition and pMTD, and this inhibition is mediated by a hyperactive “behavioral inhibition system (BIS)” composed of limbic system structures (and associated prefrontal connections). This case study used functional magnetic resonance imaging to detect brain activation changes associated with successful management of pMTD, thereby evaluating possible neural correlates of this poorly understood disorder. A 61-year-old woman with moderate-to-severe pMTD underwent functional magnetic resonance imaging scans before and immediately after successful treatment using manual circumlaryngeal techniques. Experimental stimuli were blocks of repeated vowel production and overt sentence reading. Significantly greater activation was observed pre- versus posttreatment in all regions of interest during sentence production, that is, periaqueductal gray, amygdala, hypothalamus, anterior cingulate cortex, hippocampus, dorsolateral prefrontal cortex, Brodmann area 10, and premotor and inferior sensorimotor cortex. Our findings are compatible with overactivation of neural regions associated with the BIS (cingulate cortex, amygdala, hypothalamus, periaqueductal gray) and motor inhibition networks (eg, [pre-]supplementary motor area) along with the dorsolateral prefrontal cortex and medial prefrontal cortex. Heightened input from limbic regions combined with dysfunctional prefrontal regulation may interfere with laryngeal motor preparation, initiation, and execution thereby contributing to disordered voice in pMTD." @default.
• W2779893991 created "2018-01-05" @default.
• W2779893991 creator A5030490068 @default.
• W2779893991 creator A5031212154 @default.
• W2779893991 creator A5058248098 @default.
• W2779893991 creator A5078386450 @default.
• W2779893991 creator A5081778133 @default.
• W2779893991 creator A5085289697 @default.
• W2779893991 date "2019-03-01" @default.
• W2779893991 modified "2023-09-26" @default.
• W2779893991 title "Exploring the Neural Bases of Primary Muscle Tension Dysphonia: A Case Study Using Functional Magnetic Resonance Imaging" @default.
• W2779893991 cites W1417350467 @default.
• W2779893991 cites W1858302122 @default.
• W2779893991 cites W1875603556 @default.
• W2779893991 cites W1963923343 @default.
• W2779893991 cites W1968301882 @default.
• W2779893991 cites W1969100427 @default.
• W2779893991 cites W1980683585 @default.
• W2779893991 cites W1984057735 @default.
• W2779893991 cites W1984683602 @default.
• W2779893991 cites W2004638930 @default.
• W2779893991 cites W2007159602 @default.
• W2779893991 cites W2011175843 @default.
• W2779893991 cites W2016444868 @default.
• W2779893991 cites W2018504898 @default.
• W2779893991 cites W2028072567 @default.
• W2779893991 cites W2028409050 @default.
• W2779893991 cites W2034032113 @default.
• W2779893991 cites W2037164819 @default.
• W2779893991 cites W2040583781 @default.
• W2779893991 cites W2049987518 @default.
• W2779893991 cites W2050618347 @default.
• W2779893991 cites W2052110725 @default.
• W2779893991 cites W2053976840 @default.
• W2779893991 cites W2056387840 @default.
• W2779893991 cites W2056685102 @default.
• W2779893991 cites W2060760534 @default.
• W2779893991 cites W2065772006 @default.
• W2779893991 cites W2072170505 @default.
• W2779893991 cites W2072243056 @default.
• W2779893991 cites W2073014936 @default.
• W2779893991 cites W2076068537 @default.
• W2779893991 cites W2076274207 @default.
• W2779893991 cites W2077273488 @default.
• W2779893991 cites W2082503453 @default.
• W2779893991 cites W2090439810 @default.
• W2779893991 cites W2093762388 @default.
• W2779893991 cites W2098729076 @default.
• W2779893991 cites W2105639637 @default.
• W2779893991 cites W2109185612 @default.
• W2779893991 cites W2109292724 @default.
• W2779893991 cites W2112649554 @default.
• W2779893991 cites W2115478932 @default.
• W2779893991 cites W2116590517 @default.
• W2779893991 cites W2119879845 @default.
• W2779893991 cites W2122660499 @default.
• W2779893991 cites W2142106331 @default.
• W2779893991 cites W2143096004 @default.
• W2779893991 cites W2146862351 @default.
• W2779893991 cites W2151137320 @default.
• W2779893991 cites W2153113149 @default.
• W2779893991 cites W2154668720 @default.
• W2779893991 cites W2160698122 @default.
• W2779893991 cites W2170849699 @default.
• W2779893991 cites W2301669941 @default.
• W2779893991 cites W2314365900 @default.
• W2779893991 cites W2405345309 @default.
• W2779893991 cites W2415597248 @default.
• W2779893991 cites W2532254019 @default.
• W2779893991 cites W2549219646 @default.
• W2779893991 cites W2588335022 @default.
• W2779893991 cites W2592528122 @default.
• W2779893991 cites W2607476126 @default.
• W2779893991 cites W2609099897 @default.
• W2779893991 cites W2973515423 @default.
• W2779893991 cites W4242280121 @default.
• W2779893991 doi "https://doi.org/10.1016/j.jvoice.2017.11.009" @default.
• W2779893991 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/29273230" @default.
• W2779893991 hasPublicationYear "2019" @default.
• W2779893991 type Work @default.
• W2779893991 sameAs 2779893991 @default.
• W2779893991 citedByCount "20" @default.
• W2779893991 countsByYear W27798939912019 @default.
• W2779893991 countsByYear W27798939912020 @default.
• W2779893991 countsByYear W27798939912021 @default.
• W2779893991 countsByYear W27798939912022 @default.
• W2779893991 countsByYear W27798939912023 @default.
• W2779893991 crossrefType "journal-article" @default.
• W2779893991 hasAuthorship W2779893991A5030490068 @default.
• W2779893991 hasAuthorship W2779893991A5031212154 @default.
• W2779893991 hasAuthorship W2779893991A5058248098 @default.
• W2779893991 hasAuthorship W2779893991A5078386450 @default.
• W2779893991 hasAuthorship W2779893991A5081778133 @default.
• W2779893991 hasAuthorship W2779893991A5085289697 @default.
• W2779893991 hasConcept C105702510 @default.
• W2779893991 hasConcept C140530291 @default.
• W2779893991 hasConcept C15744967 @default.
• W2779893991 hasConcept C169760540 @default.

• next