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• W2783607089 endingPage "180" @default.
• W2783607089 startingPage "180" @default.
• W2783607089 abstract "Periodontal disease is a well-regulated response to bacterial infection directed by the inflammatory cells of the host immune system. The host response to injury or insult is implicated to be a vital feature of the majority of periodontal diseases. The excessive activation of neutrophils plays a role in the pathogenesis in diseases such as acute respiratory distress syndrome, rheumatoid arthritis, and periodontitis by contributing to inflammatory tissue injury. In the recent times, there has been a shift of paradigm from a hypo- to hyper-responsive/primed model of neutrophil dysfunction in periodontal etiopathogenesis. The aim of this review is to outline the mechanisms and effects of neutrophil priming, and thereafter, discuss the current controversy that exists regarding the role of primed neutrophils in periodontal etiopathogenesis." @default.
• W2783607089 created "2018-01-26" @default.
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• W2783607089 date "2017-01-01" @default.
• W2783607089 modified "2023-10-17" @default.
• W2783607089 title "Neutrophil priming: Implications in periodontal disease" @default.
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• W2783607089 doi "https://doi.org/10.4103/jisp.jisp_385_15" @default.
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