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• W2784161421 abstract "The etiology of multiple sclerosis (MS) involves a complex interplay of genetic and environmental factors. Epidemiologic studies have furthered our understanding of these risk factors but remain limited by residual confounding and potential for reverse causation, particularly in MS where time of disease onset is not known. Mendelian randomization (MR) uses genetic variants to study the causal effect of modifiable exposures on an outcome. This method avoids some of the limitations of classical epidemiology and can strengthen causal inference. Here, we introduce the basic concepts of MR and review its contributions to the field of MS. Indeed, several studies using MR have now provided support for a causal role for low vitamin D level and obesity in the development of MS." @default.
• W2784161421 created "2018-01-26" @default.
• W2784161421 creator A5035461704 @default.
• W2784161421 creator A5074916837 @default.
• W2784161421 date "2018-01-01" @default.
• W2784161421 modified "2023-10-12" @default.
• W2784161421 title "Mendelian randomization in multiple sclerosis: A causal role for vitamin D and obesity?" @default.
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• W2784161421 doi "https://doi.org/10.1177/1352458517737373" @default.
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• W2784161421 hasPublicationYear "2018" @default.
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