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- W2784832119 abstract "Proton pump inhibitor (PPI)-induced hypomagnesemia has been recognized since 2006. Our aim was to further characterize the clinical consequences and possible mechanisms of this electrolyte disorder using 4 cases. Two men (aged 63 and 81 years) and 2 women (aged 73 and 62 years) had been using a PPI (esomeprazole, pantoprazole, omeprazole, and rabeprazole, 20-40 mg) for 1-13 years. They developed severe hypomagnesemia (magnesium, 0.30 0.28 mEq/L; reference, 1.40-2.10 mEq/L) with hypocalcemia (calcium, 6.4 1.8 mg/dL), relative hypoparathyroidism (parathyroid hormone, 43 6 pg/mL), and extremely low urinary calcium and magnesium excretion. One patient was admitted with postanoxic encephalopathy after a collapse likely caused by arrhythmia. The others had electrocardiogram abnormalities (prolonged QT interval, ST depression, and U waves). Concomitant hypokalemia (potassium, 2.8 0.1 mEq/L) was considered the trigger for these arrhythmias. Hypomagnesemiainduced kaliuresis (potassium excretion, 65 24 mEq/L) was identified as the cause of hypokalemia. This series of PPI-induced hypomagnesemia shows that this is a generic effect. It also indicates that hypomagnesemia may occur within 1 year of PPI therapy initiation and can have serious clinical consequences, likely triggered by the associated hypokalemia. A high index of suspicion is required in PPI users for unexplained hypomagnesemia, hypocalcemia, hypokalemia, or associated symptoms. Am J Kidney Dis 56:112-116. © 2010 by the National Kidney Foundation, Inc." @default.
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- W2784832119 date "2010-01-01" @default.
- W2784832119 modified "2023-09-24" @default.
- W2784832119 title "CASE REPORTS A Case Series of Proton Pump Inhibitor-Induced Hypomagnesemia" @default.
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