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- W2787758032 abstract "Acute stroke has a major effect on the cerebral vasculature with disruption of the neurovascular unit, leading to vasogenic edema. Breakdown of the blood-brain barrier (BBB) in ischemic stroke occurs in the early phases of ischemia, and is accentuated by IV treatment with recombinant tissue plasminogen activator, which increases the risk of hemorrhagic transformation and intracerebral hemorrhage.1,2 In a serendipitous observation using fluid-attenuated inversion recovery (FLAIR) MRI, gadolinium-DTPA enhancement of the CSF space overlying the ischemic tissue indicated greater stroke severity, increased age of the patient, and reperfusion injury. They called this phenomenon hyperintense acute reperfusion marker (HARM), and now Hitomi et al.3 have extended the original study to show enhancement of the structures in the eye. In addition to the endothelial blood-CNS barriers that maintain CNS homeostasis, regulate nutrition and detoxification, as well as immune cell trafficking into the brain and CSF, blood-ocular barriers (BOBs) protect the compartments of the eye.4" @default.
- W2787758032 created "2018-02-23" @default.
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- W2787758032 date "2018-02-07" @default.
- W2787758032 modified "2023-09-24" @default.
- W2787758032 title "Blood-ocular barrier leakage" @default.
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- W2787758032 doi "https://doi.org/10.1212/wnl.0000000000005131" @default.
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